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Up to 50 percent of all women over the age of 50, but even younger women have at least minimal to do with it. In women with androgenetic alopecia, hair root processes are less clear.
In some cases, however, women with clearing of hair at the apex may actually have underlying hormonal disease (androgenic alopecia). This usually goes hand in hand with so-called "masculinisation marks": Reinforced hair on the upper lip, chin, nipples, belly button, pubic area and on the thighs, as well as acne. Of course, this must be carefully medically clarified.
Characteristic of androgenetic alopecia is the reduction (so-called miniaturization) of the hair follicles, which subsequently form very small, fine and thin hairs. The growth phase (anagen phase) of the follicles is increasingly shortened. This reduction is caused by an increased activity of the androgen dihydrotestosterone (DHT), which is formed in the follicle by the 5α-reductase from testosterone.
In addition, the reduced activity of aromatase seems to be important in women. This enzyme converts androgens to estrogens. If their activity is too small, androgen excess occurs. This increase is peripheral, and only a minority of those affected have elevated levels of serum androgen and symptoms of masculinization.
The hair transplantation may be in women with pronounced androgenetic alopecia, but enough donor hair in question. In advance, a detailed consultation on possible complications, costs and achievable success should take place.